You frequently ask Dr. Google about TBI
According to an article recently reported by CNN.com, among the top 10 trending health-related questions searched on Google in the United States in 2016 were questions related traumatic brain injuries or TBI.
What is TBI?
CNN recently reported:
“TBI, or traumatic brain injury, has been linked to sports injuries, combat injuries as diagnosed in war zones and physical violence.
However, TBI also made headlines [in 2016] when cell phone video of Keith Lamont Scott being fatally shot by Charlotte police was released in November and his wife is heard in the video screaming, “He has a TBI.”
Scott’s family said the injury, caused by a motorcycle accident, resulted in him stuttering or sometimes being forgetful.
A TBI can occur when a blow or jolt to the head causes damage to the brain, which can range in severity.
Some symptoms may appear immediately while others may not appear until days or weeks following the injury.
Symptoms include headache, loss of coordination, loss of consciousness, seizures, slurred speech, weakness or numbness, dilated eye pupils or the inability to awaken from sleep.”
Here is what most people don’t know – and what wouldn’t come up in a google search as it relates to TBI. That is, until now! It’s called the BDNF gene and it is very relevant to TBI’s.
Your BDNF gene is responsible for making a very important protein in your brain known as Brain Derived Neurotrophic Factor (BDNF).
Adequate BDNF production is critical for healthy brain function – for maintaining neural plasticity, and for recovering from traumas that affect the brain. 1, 2 This is very relevant for both physical concussions and emotional trauma.
If you have a ‘low production’ version of the BDNF gene, you may be at an increased risk of poorer recovery from physical brain traumas such as concussions. You may also find that you tend to get ‘stuck’ on negative emotional stimuli – a tendency that can lead to increased neuroticism.
How do you know what version of the BDNF gene you have? It’s simple: get tested!
It turns out that intense exercise3 and hot saunas4 can help increase levels. But there is also an exciting naturally occurring ingredient that is showing tremendous promise in elevating BDNF.
Coffee is a rich source of disease-fighting antioxidants. And studies have shown that it may reduce cavities, boost athletic performance, improve moods, and stop headaches — not to mention reduce the risk of type 2 diabetes, colon cancer, liver cancer, gall stones, cirrhosis of the liver, and Parkinson’s diseases. But coffee in and of itself isn’t the solution!
Traditionally, the coffee bean has been extracted for roasting and the surrounding fruit discarded. But, the whole fruit (including the bean) of the coffee plant has the power to increase BDNF. 5
Here’s how. Recall that BDNF gene plays a central role in maintaining the survival, growth and maintenance of the cells in your brain. As mentioned, a ‘suboptimal level’ version of this gene can mean that you are more likely to be neurotic or even to be ‘stuck’ on negative memories. This gene also appears to play a role in eating, drinking and body weight. But most importantly, and as previously mentioned, this gene plays a critical role in facilitating neuronal recovery after a concussion.
The incredibly exciting news is that after years of careful clinical research, scientists discovered that ingesting small amounts of whole coffee fruit concentrate significantly increases brain derived neurotropic factor (BDNF) levels in humans.
It is important to note that coffee bean will not work for the same effect. Green coffee bean extracts (chlorogenic acids) also do not work. Drinking coffee won’t do the job either (although don’t stop drinking it since it has its own benefits).
Only the whole coffee fruit goes “beyond the bean” and delivers support for naturally declining neuroprotein levels in humans. If your genes feature a ‘suboptimal level’ version of the BDNF gene, you’ll see whole coffee fruit listed in your youtrients formula.
1 – https://www.ncbi.nlm.nih.gov/pubmed/27396498
2 – https://www.ncbi.nlm.nih.gov/pubmed/28100103
3 – https://www.ncbi.nlm.nih.gov/pubmed/28818262
4 – http://www.jneurosci.org/content/26/15/3899.long
5 – https://www.ncbi.nlm.nih.gov/pubmed/23312069